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Indolecarbinol and prostate cancer. Induction of growth arrest and apoptosis in human breast cancer cells by 3,3-diindolylmethane is associated with induction and nuclear localization of p27kip. Mol Cancer Ther ;7: NF-kappaB signalling pathway and its therapeutic implications in human diseases.

Int Rev Immunol ; Inhibition of angiogenesis and invasion by 3,3'-diindolylmethane is mediated by the nuclear factor-kappaB downstream target genes MMP-9 and uPA that regulated bioavailability of vascular endothelial growth factor in prostate cancer. Down-regulation of androgen receptor by 3,3'-diindolylmethane contributes to inhibition of cell proliferation and induction of apoptosis in both hormone-sensitive LNCaP and insensitive CB prostate cancer cells.

Role of nuclear factor-kappa B signalling in anticancer properties of indole compounds. J Exp Clin Med ;3: Cancer Prev Res ;2: J Cell Biochem ; Down-regulation of uPA and uPAR by 3,3'-diindolylmethane contributes to the inhibition of cell growth and migration of breast cancer cells. Mechanism and its regulation of tumor-induced angiogenesis. Caspase cascades in apoptosis caspases-their role in cell death and cell survival. Marek L, Henning W. Molecular Biology Intelligence Unit Hague A, Paraskeva C.

Nature Cell Death and Differentiation , September;3: Food Chem Toxicol ; J Comput Aided Mol Des ; J of Molecular Structure Improvement of cytotoxic and apoptogenic properties of crocin in cancer cell lines by its nanoliposomal form. Pharm Biol Oct;49 Bcl-2 family-mediated apoptotic effects of 3,3' diindolylmethane DIM in human breast cancer cells. Translocation of Bax to mitochondria induces apoptotic cell death in indolecarbinol I3C treated breast cancer cells.

Indolecarbinol I3C induces apoptosis in tumorigenic but not in nontumorigenic breast epithelial cells. Inactivation of akt and NF-kappaB play important roles during indolecarbinol-induced apoptosis in breast cancer cells. Gene expression profiling revealed survivin as a target of 3,3'-diindolylmethane-induced cell growth inhibition and apoptosis in breast cancer cells. Sensitization of squamous cell carcinoma to cisplatin induced killing by natural agents.

Fas-mediated apoptosis in cholangiocarcinoma cells is enhanced by 3,3'-diindolylmethane through inhibition of AKT signalling and FLICE-like inhibitory protein. Am J Pathol ; Comparison of growth inhibition profiles and mechanisms of apoptosis induction in human colon cancer cell lines by isothiocyanates and indoles from Brassicaceae.

Activation of caspase-8 contributes to 3,3'-Diindolylmethane-induced apoptosis in colon cancer cells. Inhibition of proliferation of a colon cancer cell line by indolecarbinol.

Indolecarbinol inhibits the growth of human colon carcinoma cells but enhances the tumour multiplicity and volume of azoxymethane-induced rat colon carcinogenesis. Int J Oncol ; Multiple, disparate roles for calcium signalling in apoptosis of human prostate and cervical cancer cells exposed to diindolylmethane.

Mol Cancer Ther ;5: Mechanisms of tumour modulation by indolecarbinol. Disposition and excretion in male Fischer rats. Drug Metab Dispos ; The effect of indolecarbinol and sulforaphane on a prostate cancer cell line.

ANZ J Surg ; Indolecarbinol I3C induced cell growth inhibition, G1 cell cycle arrest and apoptosis in prostate cancer cells. Plant-derived 3,3'-Diindolylmethane is a strong androgen antagonist in human prostate cancer cells. Drug Intel Clin Pharm ; Cancer Treatment Rep ; Labelled compounds of interest as antitumor agents—VIII. J Label Compd Radiopharm ; Indole derivatives as anticancer agents. United States Patent Application Publication, pub. Synthesis and preliminary cytotoxic evaluation of substituted indoles as potential anticancer agents.

Chinese Chemical Letters ; Synthesis and Evaluation of 3-Aroylindoles as Anticancer Agents: J Med Chem ; Opportunity, challenge and scope of natural products in medicinal chemistry. Chapter 7, Marine natural alkaloids as anticancer agents; p. Aust J Chem ; Can J Chem ; Acta Pharm Sinica ;